Basic knowledge


Autoimmune diseases and multiple sclerosis

Autoimmune diseases

Autoimmune diseases are steadily on the rise and are now the third most common disease after cardiovascular and tumor diseases in Switzerland. There are 80 to 100 different autoimmune diseases, one of which is multiple sclerosis. 
An autoimmune disease leads to a malfunction of the immune system, where the body’s defense mechanisms falsely attack the body’s own tissues, cells and organs, causing chronic inflammation.

How do autoimmune diseases develop?

Special white blood cells called T-lymphocytes play a key role in the immune system. These blood cells undergo an ‘education’ in the thymus during childhood, in which they learn to distinguish the body’s own structures from foreign bodies. This leads to the formation of an immune tolerance: the immune system is ‘tolerant’ of its own body structures and recognizes these as ‘body’s own’. Whereas a healthy organism disposes of any misdirected T-lymphocytes that occur, with an autoimmune disease, the body can no longer, or only insufficiently, dispose of the misdirected T-lymphocytes, and the defense mechanism is directed against the body’s own tissue.

Organ-specific autoimmune diseases produce autoantibodies only against a specific organ, for example:

  • Hashimoto’s thyroiditis forms autoantibodies against thyroid tissue
  • Graves’ Disease forms autoantibodies against thyroid TSH receptors
  • Ulcerative colitis and Crohn’s disease form autoantibodies against intestinal mucosa
  • Type 1 diabetes mellitus forms autoantibodies against the pancreas
  • Myasthenia gravis forms autoantibodies against acetylcholine receptors on motor endplates
  • Multiple sclerosis forms autoantibodies against the myelin sheath of nerve fibres

Systemic autoimmune diseases target autoantibodies throughout the entire system, for example:

  • Rheumatoid arthritis (chronic polyarthritis)
  • Atherosclerosis
  • Systemic lupus erythematosus (SLE) – reactions against numerous organs
  • Polymyositis – inflammation of the muscles or associated tissues
  • Sjögren syndrome (SjS, SS) – autoantibodies against exocrine glands
  • Scleroderma (systemic sclerosis) – changes to connective tissues of the skin, vessels and internal organs


Despite intensive research, the exact causes of autoimmune diseases are still not sufficiently understood. We assume that it is only through the interaction of various factors that a disruption of the immunological balance can occur and thus trigger an autoimmune process. Among the causes we count:

  • External factors:
    • Viruses, bacteria, parasites
    • Environmental pollutants such as heavy metals, xenohormones including plastics, pesticides, insecticides, plasticizers, etc.
    • Medicines, vaccinations
    • Chronic inflammatory processes in the tooth and jaw area
    • Stress
    • Nicotine
  • Genetic factors (HLA-associated diseases such as ankylosing spondylitis)
  • Concomitant cancers
  • Pregnancy (during pregnancy there is an exceptional immunological situation)
  • Impaired regulation of the immune system, such as food intolerance, leaky gut, allergies

Our approach for autoimmune diseases

Through extensive diagnostics, we find out which disruptive factors affect the immune system. For this, we work with:

  • Laboratory diagnostics, including autoantibodies, inflammatory parameters, viral/parasite exposure, heavy metal exposure, xenohormones, intestinal check, food intolerances, allergies, and others
  • Thermography to detect blockages
  • Heart rate variability (HRV) to determine the current stress situation
  • Panoramic view of the teeth and dental consultation to rule out inflammatory tooth-jaw processes
  • Darkfield to assess the inner environment
  • Global Diagnostic (vital field measurement)


Our therapeutic approach for autoimmune diseases

Our treatment approaches for autoimmune processes are long-term and depend on test results. Since autoimmune processes are influenced by several factors, different therapies are used:

  • Colon hydrotherapy with liver detoxification
  • DMPS and lead infusions, plus oral medication
  • Infusion therapy (for oxidative/nitrosative stress, mitochondrial diseases, vitamin deficiencies, etc.)
  • Neural therapy
  • Hyperthermia
  • Dental restoration
  • Diet change after testing


Multiple sclerosis (MS)

Organ-specific autoimmune diseases include multiple sclerosis (MS), which is also known as encephalomyelitis disseminata. MS is characterized by chronic inflammation of the central nervous system (CNS) in the brain and spinal cord. The inflammation leads to a partial destruction of the medullary sheath, also called the myelin sheath. This consists of fats and proteins that wrap around the nerve fibres like an insulating layer. It protects the nerve fibre, ensuring its nutrition and the rapid transmission of nerve impulses through the spinal cord into the rest of the body. Not only the myelin sheath, but also the nerve fibres themselves can be damaged to varying degrees. Then they can no longer pass the electrical impulses between the different nerve fibres and the body cells, such as muscle cells, without interference.

That leads to:

  • Disruption of motor skills
  • Disruption of sensations (sensors)
  • Diverse other symptoms, such as:
    • visual disturbances
    • fatigue
    • depression
    • bladder and intestinal disorders
    • reduced mental performance
    • and many more.

The course of MS can vary considerably. There is a relapsing-remitting course, in which the disease state remains the same between episodes. During an episode, however, the existing symptoms worsen. Alternatively, there is a chronically progressive course. In the case of a primary chronically progressive course, there is a gradual worsening of the symptoms from the outset. A secondary chronically progressive course can occur after a prolonged period of relapsing. It is not for no reason that MS is called the disease with a thousand faces.

We know from experience that more women than men develop MS and the onset of the disease is more prevalent in early adulthood between the ages of 20 and 40 years. As a rule, patients already come to us with the diagnosis of MS and want to know how they can stop the disease and improve their general condition.

Our approach for multiple sclerosis

Together with the patient, we find out which of the following disturbance and risk factors are present and how we can positively influence them:

  • Increased viral load, especially Epstein-Barr virus (EBV), herpes virus
  • Bacteria, particularly chlamydia
  • Heavy metals, especially neurotoxic metals such as mercury, lead, cadmium, arsenic
  • Xenohormones such as plastics, pesticides, insecticides, plasticizers, etc.
  • Disturbed detoxification function (Gen Panel Detox)
  • Disruptive dental materials such as amalgam, gold, titanium and others, and/or chronic inflammatory processes such as, neuralgia-inducing cavitational osteonecrosis (NICO)
  • Medicines, vaccinations
  • Nitrosative stress/mitochondrial dieseases


Our therapeutic approach for multiple sclerosis

Our therapeutic approach is dependent on test results. The most commonly used therapies are:

  • Physical stabilization (synthesis, mitochondrial, phosphatidylcholine, muscle-nerve infusions, etc.), plus orthomolecular therapy
  • Detoxification and elimination: colon hydro-therapy, detoxification infusions (DMPS/EDTA/discharge infusions
  • Dental restoration
  • Physical therapies
  • Psychotherapeutic intervention
  • Neural therapy (pain therapy/interference therapy)
  • Hyperthermia (to reduce viral load, stimulate detoxification)
  • Nutrition therapy

It is important to us to support patients with MS therapeutically in combination with the treating neurologist in the long term. Our goal is to halt disease progression, stabilize general condition and improve quality of life by eliminating disruptive factors and risk factors.